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However, it has not always been know as Parkinson's disease.
“And then half of those participants, or nearly one quarter of all participants with Parkinson’s, were really having a difficult time consistently with their memory, enough that it would be noticeable to other people,” said Dr. Tanner, adding that researchers were encouraged by the fact that most participants in the initial stages of Parkinson’s were not having significant memory problems.
Launched in January 2013, the NINDS Parkinson’s Disease Biomarkers Program (PDBP) aims to accelerate the search for biomarkers through eleven research projects. Seven of the research sites also collect clinical samples using a standardized methodology for the collection of blood, DNA, and cerebral spinal fluid (CSF).
That disease is Parkinson’s disease.
However, for a patient of Parkinson’s Disease who is diagnosed on average at the age of 50, these every day activities take a great deal of time, attention, and effort to be preformed (Huston)....
Any somatic and genetic variation in the brain can lead to various pathological and degenerative conditions like Alzheimer’s disease, Parkinson’s disease, Pick’s disease, Multiple Sclerosis etc.
Parkinson disease is their affliction....
In addition, being male, an older age at onset, presentation of a rigid/hypokinetic type of the disorder (rather than tremor-predominant disease), the presence of co-morbidities, and early features of postural instability or gait disorder are all features that predict a more rapidly progressing disease.
Recent practice parameters described the following features that might point to a diagnosis of parkinsonism associated with another disorder, rather than idiopathic Parkinson's disease:
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In the year 1991, he was diagnosed with Parkinson’s disease.
It was in the 1960s that that the chemical differences in the brains of Parkinson's patients were identified. The low levels of dopamine cause the degeneration of nerve cells in part of the brain called the . It was this discovery that lead to the first effective medicinal treatment of the disease. In the 1960s the drug Levodopa was first administered to treat the symptoms and has since become the "gold standard" in medication.
Parkinson's disease is characterised by three cardinal symptoms:
Since the 1960's research has continued to progress at a rapid rate. Despite the fact there is still no cure, the symptoms can now be effectively controlled and reduced in severity. The Parkinson's Disease Foundation was established in America in1957 to assist sufferers and to fund and promote further research. Many other foundations assisting the cause have been established in the following years. A notable recent addition is the Michael J Fox Foundation, named after the much loved television and movie actor. The foundation has been very public about its goal of developing a cure for the disease within this decade. Since its inception in 2000 it has succeeded in raising over 90 million US dollars.
Parkinson disease has no antidote but has altered treatments.
The olfactory system is used by the body to detect smell. It is now well established that Parkinson's sufferers have impaired function of this system. Therefore testing this system can be an indicator of Parkinsonism. It usually involves the patient smelling a variety of odors, and then making a choice from a variety of possible answers for each one.
Parkinson's Disease is a common neurologic disorder for the elderly.
The important thing to realize about Parkinson's Disease is that there is no single cause method to make a positive Parkinson's Disease diagnosis. Advancements in technology have made Parkinson's Disease diagnosis more accurate. With technology like neuronimaging, one can see in 3D the damage that may be occurring in a patient's brain. Genetic testing makes it possible to view the disease at a molecular level. It can be frustrating to go through many different types of tests but it is important that your physician makes the correct diagnosis.
Health Essays - Parkinson's Disease - UK Essays | UKEssays
There are currently four on the cause of Parkinson's Disease: (a) Oxidation of molecules such as iron in the Substantia Nigra by free radicals kill the dopaminergic neurons (1); (b) environmental toxins similar to MPTP (a compound related to the painkiller Demerol), which have been shown to cause Parkinsonism-like symptoms, kill the neurons (1,2); (c) a mysteriously increased rate of dopaminergic neuronal apoptosis in some individuals (1); and (d) Genetic influence, as the penetrance of PD has been observed at higher levels within some families (1).
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